Lifespan maxxers, how do you keep your arteries unclogged?

Lifespan maxxers, how do you keep your arteries unclogged?

  1. 4 days ago
    Anonymous

    i chug 2 cups of drain cleaner every morning

    • 4 days ago
      Anonymous

      just did this and I died. what could I have done wrong to get that effect instead?

      • 4 days ago
        Anonymous

        Did you plug your anus giving it time to act before draining?

    • 3 days ago
      Anonymous

      just did this and I died. what could I have done wrong to get that effect instead?

      retards
      you've gotta inject it

  2. 4 days ago
    Anonymous

    Minimise inflammation.
    Minimise / avoid seed oils.
    Minimise / avoid added/refined sugars/
    If you drink milk, drink Casein A2 milk from Jersey cows or sheep/goats.
    If you eat meat, eat grass-fed ruminants (beef/lamb etc)
    Eat in-season vegetables.
    Consume HIGH QUALITY olive oil. Buy a reputable brand that has been tested for provenance.
    Take gastro-coated aspirin tablets every day.
    MINIMISE INFLAMMATION.
    Don't smoke.
    Don't be diabetic.
    Walk 15,000 steps every day.
    Fast 24 hours at least once a month.

    Heart disease is the result of chronic vascular inflammation + damage. When your blood vessel linings are damaged, clots form to repair the damage. If these clotting processes (thromogenesis) occur faster than the repair + breakdown processes (thrombolysis) then the clots will keep growing ("arterial plaques").

    • 4 days ago
      Anonymous

      >INFLAMMATION
      What does this mean? Don't prick your nose or get dirt under your nails? Or are you talking about the inflammation values?

      • 4 days ago
        Anonymous

        I mean the immune system response to non-self antigens. Poor gut health leads to leaky-gut syndrome which leads to bacteria and inflammatory food proteins entering the blood stream and increasing systemic inflammation. High blood sugar destroys the glycocalyx, the sugar-protein structures that line the inside of your blood vessels, causing inflammation.
        Smoking introduces inflammatory chemicals directly into your bloodstream via your lungs.

        • 4 days ago
          Anonymous

          high blood sugar is genetic
          there are ppl eating 0 carbs and getting pre diabetic fasting blood glucose levels

          • 4 days ago
            Anonymous

            And there are people with genetic hypercholesterolemia, it doesn't mean everyone with high serum cholesterol has it from genetic causes.

            • 4 days ago
              Anonymous

              no shit
              but avoiding sugar or even carbs in general won't do much to your blood sugar because your blood sugar needs to be in a certain range for you to not die (doesn't matter if your blood sugar spikes after eating what matters is the value average throughout the day)
              there are plenty of ppl eating even ketogenic diets and their blood sugar shows up as normal when the norm is eating a ton of carbs

              so what can we do? just lower your blood lipids
              with diet or drugs if necessary

              • 4 days ago
                Anonymous

                In 20 years you will be saying that you always knew serum cholesterol wasn't the cause of heart disease.
                Read the book. You can find a free e-book version online easily enough.

                https://i.imgur.com/IDDuQbH.png

                Minimise inflammation.
                Minimise / avoid seed oils.
                Minimise / avoid added/refined sugars/
                If you drink milk, drink Casein A2 milk from Jersey cows or sheep/goats.
                If you eat meat, eat grass-fed ruminants (beef/lamb etc)
                Eat in-season vegetables.
                Consume HIGH QUALITY olive oil. Buy a reputable brand that has been tested for provenance.
                Take gastro-coated aspirin tablets every day.
                MINIMISE INFLAMMATION.
                Don't smoke.
                Don't be diabetic.
                Walk 15,000 steps every day.
                Fast 24 hours at least once a month.

                Heart disease is the result of chronic vascular inflammation + damage. When your blood vessel linings are damaged, clots form to repair the damage. If these clotting processes (thromogenesis) occur faster than the repair + breakdown processes (thrombolysis) then the clots will keep growing ("arterial plaques").

              • 4 days ago
                Anonymous

                we've known the cause of atherosclerosis
                for 60 years now
                this thread is discussing atherosclerosis specifically not heart disease in general which includes genetic defects and early development malformations
                the cause of atherosclerosis is high blood lipids

              • 4 days ago
                Anonymous

                Read the book then. If you're so sure that the evidence is that strong, then it won't change your mind.
                The science is not settled. Science is never settled. There are a lot of cardiologists sceptical of the cholesterol hypothesis. That isn't to say that serum cholesterol isn't a factor or that it isn't involved, or that a high cholesterol level might worsen it, but it is NOT the underlying cause. Just like the smoke in a house fire might be what finally kills you, rather than the fire itself or the lack of oxygen, high serum cholesterol can work in tandem with other risk factors to worsen your health.
                https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4086917/

              • 4 days ago
                Anonymous

                >but avoiding sugar or even carbs in general won't do much to your blood sugar
                Didn't read further. You have no idea what you are talking about.

              • 4 days ago
                Anonymous

                you certainly don't

              • 4 days ago
                Anonymous

                >no u
                Ok buddy

        • 4 days ago
          Anonymous

          "Leaky gut syndrome" does not exist. Also, bacteria entering the bloodstream through the gut? You can't seriously think people will get bacteremic just by eating regular food.

    • 4 days ago
      Anonymous

      What's the deal with aspirin? I thought that taking it daily will mess up with your lining

      • 4 days ago
        Anonymous

        >I thought that taking it daily will mess up with your lining
        It can if you take uncoated aspirin. Aspirin is bad for your stomach lining but it doesn't cause issues for your intestinal lining, whereas ibuprofen/paracetamol are OK for your stomach lining but are bad for your intestinal lining.
        Gastro-enteric coated aspirin tablets won't dissolve until they reach your small intestine, protecting your stomach lining.

      • 4 days ago
        Anonymous

        aspirin is also toxic for the inner ear
        tinnitus-bros beware
        if you take otc oral (analgin, paracetamol, the likes...) painkillers too much or too frequent
        youre gonna get the nastiest fucking screech you never thought of
        All NSAIDS are toxic for your ears
        dont eat it just cause some schmuck told you to
        if youre in pain for too long either use painkiller steroids, opiods, or deal with it somehow
        if youre not, why the fuck are you stuffing pills down your throat

    • 4 days ago
      Anonymous

      >Minimise / avoid added/refined sugars/
      >MINIMISE INFLAMMATION.
      Pick one. The main way of counteracting inflammation is with sugar.

      • 4 days ago
        Anonymous

        >The main way of counteracting inflammation is with sugar.
        Lol
        lmao, even

      • 4 days ago
        Anonymous

        Eat fruits. He said refined sugar

      • 4 days ago
        Anonymous

        Your body produces sugar you dumb cunt. You ever heard of gluconeogenesis?

        • 4 days ago
          Anonymous

          How is that relevant to his point? Is your point that if your body makes it it isn't beneficial to consume?

        • 4 days ago
          Anonymous

          And that process is inherently stressful. Hence you need sugar to give your body some reprieve.

    • 4 days ago
      Anonymous

      https://drmalcolmkendrick.org/

    • 4 days ago
      Anonymous

      Schizophrenic ranting. Animal fat clogs your arteries.

      • 4 days ago
        Anonymous

        >Animal fat clogs your arteries.
        that certainly explains why everyone who ate beef stews every day from 1AD until 1900 died of something unrelated to their heart at like 82

        • 3 days ago
          Anonymous

          To be fair historically most people didn't eat meat every day

      • 4 days ago
        Anonymous

        Myth. look up the French paradox.

    • 4 days ago
      Anonymous

      Why do I suspect that you only started avoiding seed oils this year?

      • 4 days ago
        Anonymous

        3 years ago actually.

      • 4 days ago
        Anonymous

        How does someone even get seed oils in their diet if they are eating real food and not processed junk, seems like anyone lifting would have a seed oil free diet

        • 4 days ago
          Anonymous

          i mix olive oil and seasonings and rub a thin coat on my chicken breast before baking. am i gonna die chads?

          • 4 days ago
            Anonymous

            Your fine

        • 4 days ago
          Anonymous

          seed oils in everything

          • 4 days ago
            Anonymous
  3. 4 days ago
    Anonymous

    keep your LDL-C below 70mg/dl

  4. 4 days ago
    Anonymous

    Fish oil (>600mg) and NAC (1.2g twice a day)

    • 4 days ago
      Anonymous

      https://i.imgur.com/IDDuQbH.png

      Minimise inflammation.
      Minimise / avoid seed oils.
      Minimise / avoid added/refined sugars/
      If you drink milk, drink Casein A2 milk from Jersey cows or sheep/goats.
      If you eat meat, eat grass-fed ruminants (beef/lamb etc)
      Eat in-season vegetables.
      Consume HIGH QUALITY olive oil. Buy a reputable brand that has been tested for provenance.
      Take gastro-coated aspirin tablets every day.
      MINIMISE INFLAMMATION.
      Don't smoke.
      Don't be diabetic.
      Walk 15,000 steps every day.
      Fast 24 hours at least once a month.

      Heart disease is the result of chronic vascular inflammation + damage. When your blood vessel linings are damaged, clots form to repair the damage. If these clotting processes (thromogenesis) occur faster than the repair + breakdown processes (thrombolysis) then the clots will keep growing ("arterial plaques").

      none of this shit will do anything

      as long as your LDL-C is high you will still get soft plaque blockage

      • 4 days ago
        Anonymous

        LDL-C is a symptom. Not a cause. Smoke doesn't cause fires, and LDL-C does not cause atheroscleroris, rather it is indicative of the underlying causes of atherosclerosis.

        • 4 days ago
          Anonymous
          • 4 days ago
            Anonymous

            I eat over 200g of carbs every day. Oats + fruits are virtually the only

        • 4 days ago
          Anonymous

          wrong
          blood lipids get stuck in your arterial walls no matter
          to avoid it you need to have low blood lipids specifically your LDL-C in order for the blockage to be minimal
          nothing else will work

          atherosclerosis is the symptom and high blood lipids are the cause
          LDL-C isn't bad it's having too much of it that is bad

          • 4 days ago
            Anonymous

            People with exceptionally low LDL-C also develop atherosclerosis. If LDL-C were the essential cause, then how do you explain these cases?
            There is no dose-dependent relationship between LDL-C levels and severity of atherosclerosis.If LDL-C were the essential cause, you would expect higher LDL-C to mean more severe atherosclerosis, which is not always the case.

            • 4 days ago
              Anonymous

              >People with exceptionally low LDL-C also develop atherosclerosis
              they don't develop it with low LDL-C they accumulate it over years and then when they are diagnosed with cardiovascular problems or hypercholesterolemia they attempt to lower their LDL-C but artery blockage takes years to reverse

              we know what happens when an animal has a high LDL-C no matter the "cause" the result is the same no matter the species, atherosclerosis.

              • 4 days ago
                Anonymous

                Yes anon, and smoke causes fires, and guns kill people.

              • 4 days ago
                Anonymous

                Read the book then. If you're so sure that the evidence is that strong, then it won't change your mind.
                The science is not settled. Science is never settled. There are a lot of cardiologists sceptical of the cholesterol hypothesis. That isn't to say that serum cholesterol isn't a factor or that it isn't involved, or that a high cholesterol level might worsen it, but it is NOT the underlying cause. Just like the smoke in a house fire might be what finally kills you, rather than the fire itself or the lack of oxygen, high serum cholesterol can work in tandem with other risk factors to worsen your health.
                https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4086917/

                ancel keys lived to 100 years old putting his own hypothesis to the test
                no one who has tried to refute him has yet reached that age

              • 4 days ago
                Anonymous

                My grandfather lived until his mid 90s on a steady diet of beef, eggs and lamb. Individual cases prove nothing. Read the book. You're obviously interested in the subject. If nothing else, it will give you a new perspective.

              • 4 days ago
                Anonymous

                I don't care about the anecdote of an online stranger who is clearly emotionally invested in trying to debunk common knowledge in a silly attempt of contrarian rebellion
                show me a single person who has ever tried to refute the blood lipid cause of atherosclerosis who has reached 100y of age
                let's see some big names and their own results

              • 4 days ago
                Anonymous

                >hahaha if you haven't died at 100yo then your theory is wrong
                Up until now I believed you were discussing the issue with me in good faith. You've descended into a logical hole with no escape.
                Let's talk about the Minnesota Coronary Experiment.

                >In 1968, about ten years after the beginning and two years after the first publication of the results of the Seven Countries Study, Keys and Ivan Frantz initiated a large randomized control trial, replacing saturated fats by food items with naturally high or artificially raised content of linoleic acid in an intervention group.
                >The study shows no positive effects of the altered dietary intake.
                >The 2016 paper "Re-evaluation of the traditional diet-heart hypothesis: analysis of recovered data from Minnesota Coronary Experiment" concludes:
                >Results – The intervention group had significant reduction in serum cholesterol compared with controls (mean change from baseline −13.8% v −1.0%; P<0.001). Kaplan Meier graphs showed no mortality benefit for the intervention group in the full randomized cohort or for any prespecified subgroup. There was a 22% higher risk of death for each 30 mg/dL (0.78 mmol/L) reduction in serum cholesterol in covariate adjusted Cox regression models (hazard ratio 1.22, 95% confidence interval 1.14 to 1.32; P<0.001). There was no evidence of benefit in the intervention group for coronary atherosclerosis or myocardial infarcts. Systematic review identified five randomized controlled trials for inclusion (n=10808). In meta-analyses, these cholesterol lowering interventions showed no evidence of benefit on mortality from coronary heart disease (1.13, 0.83 to 1.54) or all cause mortality (1.07, 0.90 to 1.27).
                Conclusions – Available evidence from randomized controlled trials shows that replacement of saturated fat in the diet with linoleic acid effectively lowers serum cholesterol but does not support the hypothesis that this translates to a lower risk of death from coronary heart disease or all causes. Findings from the Minnesota Coronary Experiment add to growing evidence that incomplete publication has contributed to overestimation of the benefits of replacing saturated fat with vegetable oils rich in linoleic acid.

                >The intervention group had significant reduction in serum cholesterol compared with controls
                >aplan Meier graphs showed no mortality benefit for the intervention group in the full randomized cohort or for any prespecified subgroup.
                >There was a 22% higher risk of death for each 30 mg/dL (0.78 mmol/L) reduction in serum cholesterol
                > In meta-analyses, these cholesterol lowering interventions showed no evidence of benefit on mortality from coronary heart disease (1.13, 0.83 to 1.54) or all cause mortality (1.07, 0.90 to 1.27).
                If serum lipoproteins are the sole cause of heart disease, why do the RCTs show no reduction in heart disease OR all-cause mortality?

              • 4 days ago
                Anonymous

                lyon diet heart is also fun, virtually identical LDL between groups but massive benefit from eating mediterranean instead of AHA slop
                no doubt from the lower amount of linoleic acid, which also means keys was double dipping by moving to the land of olive oil instead of staying and drinking basedbeans

              • 4 days ago
                Anonymous

                >In 1968, about ten years after the beginning and two years after the first publication of the results of the Seven Countries Study, Keys and Ivan Frantz initiated a large randomized control trial, replacing saturated fats by food items with naturally high or artificially raised content of linoleic acid in an intervention group.
                >The study shows no positive effects of the altered dietary intake.
                >The 2016 paper "Re-evaluation of the traditional diet-heart hypothesis: analysis of recovered data from Minnesota Coronary Experiment" concludes:
                >Results – The intervention group had significant reduction in serum cholesterol compared with controls (mean change from baseline −13.8% v −1.0%; P<0.001). Kaplan Meier graphs showed no mortality benefit for the intervention group in the full randomized cohort or for any prespecified subgroup. There was a 22% higher risk of death for each 30 mg/dL (0.78 mmol/L) reduction in serum cholesterol in covariate adjusted Cox regression models (hazard ratio 1.22, 95% confidence interval 1.14 to 1.32; P<0.001). There was no evidence of benefit in the intervention group for coronary atherosclerosis or myocardial infarcts. Systematic review identified five randomized controlled trials for inclusion (n=10808). In meta-analyses, these cholesterol lowering interventions showed no evidence of benefit on mortality from coronary heart disease (1.13, 0.83 to 1.54) or all cause mortality (1.07, 0.90 to 1.27).
                Conclusions – Available evidence from randomized controlled trials shows that replacement of saturated fat in the diet with linoleic acid effectively lowers serum cholesterol but does not support the hypothesis that this translates to a lower risk of death from coronary heart disease or all causes. Findings from the Minnesota Coronary Experiment add to growing evidence that incomplete publication has contributed to overestimation of the benefits of replacing saturated fat with vegetable oils rich in linoleic acid.

              • 4 days ago
                Anonymous

                And the oldest woman to ever live smoked every day. Who cares

              • 4 days ago
                Anonymous

                Same as that old ass black WWII veteran drank whiskey, smoked cigars, and ate red meat every day, lived to like 112 or some shit despite blacks being far more prone to cardiovascular problems.

              • 4 days ago
                Anonymous

                I dont need another identical point to reinforce my point I think it was pretty clear

              • 4 days ago
                Anonymous

                >get dunked on
                >respond with gay anecdote
                how scientific of you

              • 4 days ago
                Anonymous

                > accumulate it over years and then when they are diagnosed with cardiovascular problems or hypercholesterolemia they attempt to lower their LDL-C but artery blockage takes years to reverse
                Cope
                >we know what happens when an animal has a high LDL-C no matter the "cause" the result is the same no matter the species, atherosclerosis.
                Explain how Paul Saladino has 300 nmol/dL LDL-C and has no signs of atherosclerosis. Or how the blatant data manipulation in the Framingham study serves is proof of anything other than how desperate modern “scientists” are to prove their retarded theory that your body just clogs it’s own arteries for fun

              • 4 days ago
                Anonymous

                >Explain how Paul Saladino has 300 nmol/dL LDL-C and has no signs of atherosclerosis.

                "signs" of atherosclerosis?
                how much soft non calcified plaque foes he have on his arteries?
                talk to me when he reaches 100 years old like ancel keys did

              • 4 days ago
                Anonymous

                >pretends to be expert on Mongolian basket weaving forum
                LDL is a surrogate marker for a clinical outcome, it is not the clinical outcome and will never be the clinical outcome.
                stop posting and consider taking a basic scientific literature course before attempting to enter these discussions again

            • 4 days ago
              Anonymous

              ahh is this why it's bad to blast roids for too long?

        • 3 days ago
          Anonymous

          >smoke doesn't cause fires
          you don't mind if i use this?

          • 3 days ago
            Anonymous

            Only Gold Account™ users are allowed to repost from 4chon, anon.

  5. 4 days ago
    Anonymous

    small amount of exercise multiple times a day to keep releasing nitric oxide

  6. 4 days ago
    Anonymous

    regularly prolonged fasting is my best shot to unfucked shits

  7. 4 days ago
    Anonymous

    Vitamin K2, fiber, cardio, and 3-5 day monthly fasting are the healthiest means. Your arteries don't usually start clogging until you already have underlying damage. You can accrue damage with no warning though. So you should generally aim to keep your cholesterol profile in check through those means. Some will argue that lowering cholesterol also leads to lowering of testosterone since testosterone is derived from cholesterol. You'll have to make the judgement for yourself.
    There are other methods doctors will recommend like seed oils, but they work by oxidizing lipids. Which is good for temporary cardiac health but also leads to cancer. Many people also report feeling awful when they introduce them into their diet. So it's a balancing act and you need to decide for yourself which sounds scarier.

    • 4 days ago
      Anonymous

      Oh yeah, and Omega-3's through having fish a couple times a week.

    • 4 days ago
      Anonymous

      >Your arteries don't usually start clogging until you already have underlying damage
      the atherosclerosis process starts before puberty

      • 4 days ago
        Anonymous

        I said nothing about age. Like I said it's anyone with already existing damage. Sometimes that includes teens. https://www.healthline.com/health/heart-disease/atherosclerosis-when-it-starts#causes

        • 4 days ago
          Anonymous

          >Your arteries don't usually start clogging until you already have underlying damage
          the atherosclerosis process starts before puberty

          we've known the cause of atherosclerosis
          for 60 years now
          this thread is discussing atherosclerosis specifically not heart disease in general which includes genetic defects and early development malformations
          the cause of atherosclerosis is high blood lipids

          high blood lipids don't cause atherosclerosis
          vascular shear stress and flow turbulence is the underlying cause that creates dysfunctional endothelium.
          You would understand this if you understood what's happening at a cell and molecular level in the arteries, and thus why atherosclerosis only happens principally in the coronaries, and then in marginally less of the population in the carotids, superficial femoral, popliteal, and peroneal arteries. There is no radial artery, ulnar, brachial, etc. pathologies unless your genetics are fucked because the flow conditions aren't present there.

          one of two pathophysiologies happens:
          >fibrotic route
          1. high wall shear stress and turbulent flow induce endothelial morphologic changes
          2. vSMC's proliferate which turns into adaptive intimal thickening (AIT)
          3. adaptive intimal thickening turns into pathologic intimal thickening as a feedback loop is created where stenosis creates high shear stress which outpaces the artery's ability to dilate, which causes further vSMC proliferation
          4. this turns into a fibrotic plaque, which eventually encounters levels of shear stress sufficient to dehisce the endothelium from the smooth muscle, causing a plaque erosion
          5. Additionally, it may progress to the below route if it gets thick enough to present a hypoxic core which releases HIF-1a and VEGF

          >TCFA route
          1. high wall shear stress, turbulent flow, or low wall shear stress (bifurcations) induce endothelial dysfunction by separating VE-Cadherin tight junctions
          2. VE-cadherin separation causes the release of its anchoring molecule, p-120 catenin, which induces gene expression changes in EC's
          3. because tight junctions are severed, blood products such as lipid, sugars, cholesterol, and lysed cell products are allowed to leak into the media of the vessel
          4. These molecules do two things: accumulate in numbers, and cause vSMC's and EC's to signal inflammation using VCAM, ICAM-1, and other cytokines

          cont.

          • 4 days ago
            Anonymous

            5. VCAM, ICAM, and inflammatory load cause circulating monocytes to adhere and extravasate into the vessel wall
            6. these monocytes become macrophages and start to phagocytose the sugar, lipid, and cholesterol
            7. They become "foamy macrophages" which are just WBCs that are too fat to return to circulation
            8. In addition, by now the plaque is usually large enough to have a hypoxic center, which causes VEGF and HIF-1a release
            9. In tandem, foamy macrophages start to die, increasing the inflammatory condition of the plaque (necrotic core), while more macrophages enter both to scavenge the dead cell parts as well as build new blood vessels into the plaque
            10. the blood vessels built into the plaque are leaky and immature, which allows red blood cells to enter and die, releasing hemosiderin and causing CD163 macrophages to enter to scavenge the hemoglobin-haptoglobin complexes within the plaque
            11. eventually the plaque is too large to maintain and shear stress causes the thin fibrous cap (thin-capped fibroatheroma, TCFA) to rupture and thrombose

            There are also several other types of plaque morphologies with their own etiologies such as CTO's and healed ruptures, but those are the main two.
            as long as you have literally any fat and cholesterol in your bloodstream, atherosclerosis will progress. The main thing that has to happen is twofold:
            1. your vessels must keep dilating in response to oxygen demand in your heart and retain plasticity, e.g. you must exercise as well as vary your blood pressure
            2. you must have good collateralization because plaque WILL accumulate no matter what statin or ACEi or designer drug you're on because doctors believe that physiologically normal levels of blood lipid are bad. Collateralization saves lives and our ancestors were sufficiently non-sedentary that they had good collateralization de facto.
            >source: I have written four papers about this

            • 4 days ago
              Anonymous

              >collateralization
              what does this mean in laymans terms?

              • 4 days ago
                Anonymous

                two blood vessels that supply the same tissue
                so for instance, the "widowmaker" is the left anterior descending artery or LAD
                from the LAD, you have diagonal branches, D1, D2, D3, etc
                if you have good collateralization, D1 and D2 may supply the same tissue, D3 and distal LAD may supply the same tissue
                Basically good collateralization means that you may be able to have a total blockage in an artery and not have ill effects out of it

                basically: "you can get off the highway at either exit and still get to your house"

              • 4 days ago
                Anonymous

                so you're saying in this regard it's merely chance? how long does it take to remove deposits in the arteries through diet and exercise?

              • 4 days ago
                Anonymous

                >how long does it take to remove deposits in the arteries through diet and exercise?
                they can't be removed except with a rotoblator or an endarterectomy (which happens for carotids)
                arteries can dilate in response to flow conditions, the question is just "does the pace of plaque deposition outpace the artery's ability to compensate"
                you can have "50% stenosis" in an artery but still have good perfusion, because stenosis is just a measure of the cross-sectional area of the lumen vs the cross sectional area within the internal elastic lamina
                it's like back problems. Your body does a good job of managing the problem itself, as long as you don't fuck up and make it more difficult to do so. Despite this, on a long enough timeline everyone will get intervertebral disk degeneration, just like everyone will get some degree of heart disease.

              • 4 days ago
                Anonymous

                So how does blood LDL relate to atherosclerosis, exactly? And how important are time frames of elevation?

              • 4 days ago
                Anonymous

                >So how does blood LDL relate to atherosclerosis, exactly?
                blood LDL is an errant metric
                it doesn't really relate to the development of atherosclerosis unless you have LP(a), because LP(a) will bind to PDGF and a lot of other platelet-related factors
                there are plenty of people on statins who have absolutely dogshit arteries, and there are plenty of people with high LDL and only moderate, expected level CAD

                there's been a total misunderstanding as to what a "fatty plaque" or "fatty atheroma" is. Macrophages infiltrate in order to scavenge fat and cholesterol, however it's the macrophages that are driving the pathology. The macrophages don't just wander into the plaque, they bind to VCAM and PDGF and a whole host of other receptors in response to cytokine release due to inflammation.

                It's actually very similar to cancer, where the presence of immune response is actually counterproductive past a certain point. the TME is actually the best model environment for CAD

                >And how important are time frames of elevation?
                periodicity is good just like everything, but you're more or less out of control of this unless you're talking about YoY levels.

              • 4 days ago
                Anonymous

                >they bind to VCAM and PDGF and a whole host of other receptors in response to cytokine release due to inflammation
                ok, and how does this relate to cytokine response from resistance training?

              • 4 days ago
                Anonymous

                >ok, and how does this relate to cytokine response from resistance training?
                different kind of cytokines
                global inlfammatory cytokines are different from paracrine cytokines like HIF-1 and juxtacrine signals like VCAM

            • 4 days ago
              Anonymous

              Is there a way for fages to clean up the vessel without forming a plaque? How would you reccomend to clean the vessels?

            • 4 days ago
              Anonymous

              >our ancestors were sufficiently non-sedentary that they had good collateralization de facto.
              How exactly physical activity affects collateralization?

          • 4 days ago
            Anonymous

            5. VCAM, ICAM, and inflammatory load cause circulating monocytes to adhere and extravasate into the vessel wall
            6. these monocytes become macrophages and start to phagocytose the sugar, lipid, and cholesterol
            7. They become "foamy macrophages" which are just WBCs that are too fat to return to circulation
            8. In addition, by now the plaque is usually large enough to have a hypoxic center, which causes VEGF and HIF-1a release
            9. In tandem, foamy macrophages start to die, increasing the inflammatory condition of the plaque (necrotic core), while more macrophages enter both to scavenge the dead cell parts as well as build new blood vessels into the plaque
            10. the blood vessels built into the plaque are leaky and immature, which allows red blood cells to enter and die, releasing hemosiderin and causing CD163 macrophages to enter to scavenge the hemoglobin-haptoglobin complexes within the plaque
            11. eventually the plaque is too large to maintain and shear stress causes the thin fibrous cap (thin-capped fibroatheroma, TCFA) to rupture and thrombose

            There are also several other types of plaque morphologies with their own etiologies such as CTO's and healed ruptures, but those are the main two.
            as long as you have literally any fat and cholesterol in your bloodstream, atherosclerosis will progress. The main thing that has to happen is twofold:
            1. your vessels must keep dilating in response to oxygen demand in your heart and retain plasticity, e.g. you must exercise as well as vary your blood pressure
            2. you must have good collateralization because plaque WILL accumulate no matter what statin or ACEi or designer drug you're on because doctors believe that physiologically normal levels of blood lipid are bad. Collateralization saves lives and our ancestors were sufficiently non-sedentary that they had good collateralization de facto.
            >source: I have written four papers about this

            >high blood lipids don't cause atherosclerosis
            I know, I never said they did. I said they only come into play if there's preexisting damage. Can you morons read or do you just have prewritten blog posts correcting things nobody said?

            • 4 days ago
              Anonymous

              no i was agreeing with you

              • 4 days ago
                Anonymous

                Ah I'm an overly defensive gay then. My bad.

          • 4 days ago
            Anonymous

            5. VCAM, ICAM, and inflammatory load cause circulating monocytes to adhere and extravasate into the vessel wall
            6. these monocytes become macrophages and start to phagocytose the sugar, lipid, and cholesterol
            7. They become "foamy macrophages" which are just WBCs that are too fat to return to circulation
            8. In addition, by now the plaque is usually large enough to have a hypoxic center, which causes VEGF and HIF-1a release
            9. In tandem, foamy macrophages start to die, increasing the inflammatory condition of the plaque (necrotic core), while more macrophages enter both to scavenge the dead cell parts as well as build new blood vessels into the plaque
            10. the blood vessels built into the plaque are leaky and immature, which allows red blood cells to enter and die, releasing hemosiderin and causing CD163 macrophages to enter to scavenge the hemoglobin-haptoglobin complexes within the plaque
            11. eventually the plaque is too large to maintain and shear stress causes the thin fibrous cap (thin-capped fibroatheroma, TCFA) to rupture and thrombose

            There are also several other types of plaque morphologies with their own etiologies such as CTO's and healed ruptures, but those are the main two.
            as long as you have literally any fat and cholesterol in your bloodstream, atherosclerosis will progress. The main thing that has to happen is twofold:
            1. your vessels must keep dilating in response to oxygen demand in your heart and retain plasticity, e.g. you must exercise as well as vary your blood pressure
            2. you must have good collateralization because plaque WILL accumulate no matter what statin or ACEi or designer drug you're on because doctors believe that physiologically normal levels of blood lipid are bad. Collateralization saves lives and our ancestors were sufficiently non-sedentary that they had good collateralization de facto.
            >source: I have written four papers about this

            Cool tha is bro. Fug I gotta up the cardio

          • 4 days ago
            Anonymous

            5. VCAM, ICAM, and inflammatory load cause circulating monocytes to adhere and extravasate into the vessel wall
            6. these monocytes become macrophages and start to phagocytose the sugar, lipid, and cholesterol
            7. They become "foamy macrophages" which are just WBCs that are too fat to return to circulation
            8. In addition, by now the plaque is usually large enough to have a hypoxic center, which causes VEGF and HIF-1a release
            9. In tandem, foamy macrophages start to die, increasing the inflammatory condition of the plaque (necrotic core), while more macrophages enter both to scavenge the dead cell parts as well as build new blood vessels into the plaque
            10. the blood vessels built into the plaque are leaky and immature, which allows red blood cells to enter and die, releasing hemosiderin and causing CD163 macrophages to enter to scavenge the hemoglobin-haptoglobin complexes within the plaque
            11. eventually the plaque is too large to maintain and shear stress causes the thin fibrous cap (thin-capped fibroatheroma, TCFA) to rupture and thrombose

            There are also several other types of plaque morphologies with their own etiologies such as CTO's and healed ruptures, but those are the main two.
            as long as you have literally any fat and cholesterol in your bloodstream, atherosclerosis will progress. The main thing that has to happen is twofold:
            1. your vessels must keep dilating in response to oxygen demand in your heart and retain plasticity, e.g. you must exercise as well as vary your blood pressure
            2. you must have good collateralization because plaque WILL accumulate no matter what statin or ACEi or designer drug you're on because doctors believe that physiologically normal levels of blood lipid are bad. Collateralization saves lives and our ancestors were sufficiently non-sedentary that they had good collateralization de facto.
            >source: I have written four papers about this

            I am a retard but I trust you on this. What can I do to prevent ateroesclerosis? Any exercises? Food? Supplements?

            • 4 days ago
              Anonymous

              avoiding inflammation is the best way, but that's just generalized inflammatory condition. The inflammation I'm talking about is fairly localized
              a good split between cardio like cycling, rowing, or swimming and good heavy strength training seems to be the best way to keep your heart healthy without eventually dooming yourself to triathlete syndrome and having good arteries but shitty valves
              unironically it's mostly genetics
              the best thing you can do is stay healthy using the above, so that when you do have a heart attack, it doesn't kill you.
              best thing to do is just stay on top of how you feel every day as you get older. getting suddenly winded with no explanation, feeling shitty and sick without doing anything, means you should probably see a cardiologist because it could mean the difference between some light PCI stenting or POBA and some dude cracking your chest.

          • 4 days ago
            Anonymous

            5. VCAM, ICAM, and inflammatory load cause circulating monocytes to adhere and extravasate into the vessel wall
            6. these monocytes become macrophages and start to phagocytose the sugar, lipid, and cholesterol
            7. They become "foamy macrophages" which are just WBCs that are too fat to return to circulation
            8. In addition, by now the plaque is usually large enough to have a hypoxic center, which causes VEGF and HIF-1a release
            9. In tandem, foamy macrophages start to die, increasing the inflammatory condition of the plaque (necrotic core), while more macrophages enter both to scavenge the dead cell parts as well as build new blood vessels into the plaque
            10. the blood vessels built into the plaque are leaky and immature, which allows red blood cells to enter and die, releasing hemosiderin and causing CD163 macrophages to enter to scavenge the hemoglobin-haptoglobin complexes within the plaque
            11. eventually the plaque is too large to maintain and shear stress causes the thin fibrous cap (thin-capped fibroatheroma, TCFA) to rupture and thrombose

            There are also several other types of plaque morphologies with their own etiologies such as CTO's and healed ruptures, but those are the main two.
            as long as you have literally any fat and cholesterol in your bloodstream, atherosclerosis will progress. The main thing that has to happen is twofold:
            1. your vessels must keep dilating in response to oxygen demand in your heart and retain plasticity, e.g. you must exercise as well as vary your blood pressure
            2. you must have good collateralization because plaque WILL accumulate no matter what statin or ACEi or designer drug you're on because doctors believe that physiologically normal levels of blood lipid are bad. Collateralization saves lives and our ancestors were sufficiently non-sedentary that they had good collateralization de facto.
            >source: I have written four papers about this

            Beautiful

          • 4 days ago
            Anonymous

            How to lower sheer stress on the vessels?

          • 4 days ago
            Anonymous

            5. VCAM, ICAM, and inflammatory load cause circulating monocytes to adhere and extravasate into the vessel wall
            6. these monocytes become macrophages and start to phagocytose the sugar, lipid, and cholesterol
            7. They become "foamy macrophages" which are just WBCs that are too fat to return to circulation
            8. In addition, by now the plaque is usually large enough to have a hypoxic center, which causes VEGF and HIF-1a release
            9. In tandem, foamy macrophages start to die, increasing the inflammatory condition of the plaque (necrotic core), while more macrophages enter both to scavenge the dead cell parts as well as build new blood vessels into the plaque
            10. the blood vessels built into the plaque are leaky and immature, which allows red blood cells to enter and die, releasing hemosiderin and causing CD163 macrophages to enter to scavenge the hemoglobin-haptoglobin complexes within the plaque
            11. eventually the plaque is too large to maintain and shear stress causes the thin fibrous cap (thin-capped fibroatheroma, TCFA) to rupture and thrombose

            There are also several other types of plaque morphologies with their own etiologies such as CTO's and healed ruptures, but those are the main two.
            as long as you have literally any fat and cholesterol in your bloodstream, atherosclerosis will progress. The main thing that has to happen is twofold:
            1. your vessels must keep dilating in response to oxygen demand in your heart and retain plasticity, e.g. you must exercise as well as vary your blood pressure
            2. you must have good collateralization because plaque WILL accumulate no matter what statin or ACEi or designer drug you're on because doctors believe that physiologically normal levels of blood lipid are bad. Collateralization saves lives and our ancestors were sufficiently non-sedentary that they had good collateralization de facto.
            >source: I have written four papers about this

            Wow so you're saying once the plaque starts to form (and it's very easy for it to form)
            >it's over
            and it will kill the vessel completly. I don't think it works that way honestly.

  8. 4 days ago
    Anonymous

    eat a healthy diet of Chicken or Salmon, Broccoli (cooked), legumes and whole carbs like oats
    avoid refined sugars, red meats (but not all the time), drink plenty of water, lift weights and do HIIT cardio or swimming, have a hobby and enjoy life, get 8 hours of sleep a night, reduce cortisol levels, and be happy,
    maybe take and aspirin if your doctor recommends, and get a yearly/biyearly checkup
    sleep with a weighted blanket, go to church every sunday
    also have lots of sex (with real women)

    • 3 days ago
      Anonymous

      >with real woman
      enjoy dying at your 60

  9. 4 days ago
    Anonymous

    I cut back on meat and junk food drastically this year, cholesterol numbers improved

    • 4 days ago
      Anonymous

      Good for you anon. Hope you are doing well.

  10. 4 days ago
    Anonymous

    Garlic

  11. 4 days ago
    Anonymous

    By not getting the jab

  12. 4 days ago
    Anonymous

    I'm going to kill myself at 70

  13. 4 days ago
    Anonymous

    surprised no one has mentioned the cure for this yet.
    munch k2
    google it

    • 4 days ago
      Anonymous

      wtf youre actually right

  14. 4 days ago
    Anonymous

    nattokinase supplements

  15. 4 days ago
    Anonymous

    Idk I just try to eat healthy and not make fried food a big part of my diet

  16. 4 days ago
    Anonymous

    Genetics. Avoid calcium supplements as they leave calcium deposits in the arteries. Not sure why yet but it is from over supplementation of them, particularly in older people.

  17. 4 days ago
    Anonymous

    85g fat/day
    110g protein/day
    250g+ carbs/day
    50g+ fiber/day
    1:1 omega 3/6 ratio
    20% saturated 80% unsaturated

    100% vitamin reqs met
    100% mineral reqs met
    100% amino acid profile met

    Toxins minimized through reverse osmosis filters, organic, non-gmo foods, and not using toxic cooking tools like teflon pans, or incorrectly using air fryers to lower acrylamides etc.

    Still learning, but that's a good start.

    • 4 days ago
      Anonymous

      You should also avoid aluminum pans/pots

    • 4 days ago
      Anonymous

      teflon is okay if not damaged & easy to use, buy cheap either way and replace when damaged, go for ceramic if you really dont want teflon but the non stick properties go away faster tho. also use silicone or wooden utensils & dont ever put on high fire

    • 3 days ago
      Anonymous

      What would you need carbs for? As far as I know there's nothing essential about them

  18. 4 days ago
    Anonymous

    Extended fasts. Minimum of a week. People who starve to death don't have artery plaque

  19. 4 days ago
    Anonymous

    no red meat
    no eggs
    no dairy
    no salt
    no carbs

    • 4 days ago
      Anonymous

      Its Alost like those people worked hard with their bodies all day and strengthened their arteries and heart

  20. 4 days ago
    Anonymous

    Take the antifungal pill.

    [...]

  21. 4 days ago
    Anonymous

    Vitamin K2, expresses calcium binding proteins that stops excessive blood circulation and actually puts calcium where it needs to be (in bones where it can be leeched later if the body needs it).
    Pomegranate, has a unique polyphenol that prevents NOx oxidation.
    l-citrulline, precursor for l-arginine. increases l-arginine by codependency mechanism.
    walking
    exercise

  22. 4 days ago
    Anonymous

    Avoid seed oils, poly fats.
    Eat lots of red meat, sat fats, basically eat like our ancestors did

    • 4 days ago
      Anonymous

      Avoid animal products, eat healthy vegetable oils (canola oil, sunflower oil etc)

      Avoid all kinds of fat, eat lean meat like elk, chicken breast, or rabbit. Fill in the rest of your needed calories in with carbs.

      • 4 days ago
        Anonymous

        Fat is required to live, unironically. You would die if you ate 0 fats eventually.

        • 4 days ago
          Anonymous

          >What is conversion of carbs to fat
          >What is bodyfat
          If my body could synthesize it then why would it be essential? Best part is a third of the carbs get lost as heat during the conversion process.

          • 4 days ago
            Anonymous

            You're actually braindead if you think that's how it works. Try going 0 fat for a month and let me know how you're doing, you won't since you'll be dead.

            • 4 days ago
              Anonymous

              I've gone multiple months on <20g a day. Perform way better in the gym and on cardio too. Fat is not an essential nutrient. You're thinking a white rice and fruit only diet that is indeed deadly but because of lack of protein and B12, not lack of fat.

              • 4 days ago
                Anonymous

                Got it, you're an actual braindead moron. Without dietary fat you can't absorb any fat-soluble vitamins. Thanks for confirming you're a moron vegan tho.

              • 4 days ago
                Anonymous

                I eat lean meats. I hate veganism just as much as keto for the sole reason that they push out seed oils as health food. Enjoy the insulin resistance if you combine the fat with carbs.

              • 4 days ago
                Anonymous

                ITT: retards still coping with the fact ancel keys was right all along

                you eat fat even if you don't want to it's impossible to avoid it unless you eat nothing but sugar

                >pretends to be expert on Mongolian basket weaving forum
                LDL is a surrogate marker for a clinical outcome, it is not the clinical outcome and will never be the clinical outcome.
                stop posting and consider taking a basic scientific literature course before attempting to enter these discussions again

                high blood lipids are bad in all animals and a sign of metabolic disfunction
                being myopic about muh mechanisms doesn't change the fact that having a high LDL-C is bad and it will always be bad

                >He died from eating too much meat, butter, and fat
                said no one ever

                you can't say anything when dead

                >So how does blood LDL relate to atherosclerosis, exactly?
                blood LDL is an errant metric
                it doesn't really relate to the development of atherosclerosis unless you have LP(a), because LP(a) will bind to PDGF and a lot of other platelet-related factors
                there are plenty of people on statins who have absolutely dogshit arteries, and there are plenty of people with high LDL and only moderate, expected level CAD

                there's been a total misunderstanding as to what a "fatty plaque" or "fatty atheroma" is. Macrophages infiltrate in order to scavenge fat and cholesterol, however it's the macrophages that are driving the pathology. The macrophages don't just wander into the plaque, they bind to VCAM and PDGF and a whole host of other receptors in response to cytokine release due to inflammation.

                It's actually very similar to cancer, where the presence of immune response is actually counterproductive past a certain point. the TME is actually the best model environment for CAD

                >And how important are time frames of elevation?
                periodicity is good just like everything, but you're more or less out of control of this unless you're talking about YoY levels.

                high blood lipids are bad
                you don't know what you're talking about

              • 4 days ago
                Anonymous

                >you don't know what you're talking about
                Disprove him.

              • 4 days ago
                Anonymous

                He's gonna bring up the fact that ancel keys lived to 100 again because he's still the same butthurt retard from ten hours ago.

              • 3 days ago
                Anonymous

                the fact that hypercholesterolemia is a state of disease in all animals regardless of their natural diet disproves him

                He's gonna bring up the fact that ancel keys lived to 100 again because he's still the same butthurt retard from ten hours ago.

                still no names of 100y old cholesterol denialism proponents lol

              • 3 days ago
                Anonymous

                >the fact that hypercholesterolemia is a state of disease in all animals regardless of their natural diet disproves him
                How?

              • 3 days ago
                Anonymous

                by proving that high blood lipids in the blood is not merely a symptom of disease but a state of illness in itself
                no animal gets atherosclerosis with low blood lipids and both carnivores and herbivores have low blood lipids on their natural diet unless they have some genetic fuckup

              • 4 days ago
                Anonymous

                >19 is the same as 0

  23. 4 days ago
    Anonymous

    >He died from eating too much meat, butter, and fat
    said no one ever

  24. 4 days ago
    Anonymous

    Avoid animal products, eat healthy vegetable oils (canola oil, sunflower oil etc)

    • 4 days ago
      Anonymous

      Obesity rates have gone up at the same rate seed oil consumption has. No coincidence, retard.

      • 4 days ago
        Anonymous

        >seed oils are evil
        I see you're jumping on the latest internet fad and ignoring the decades of science that shows vegetables oils are a much healthier alternative to animal fats

        • 4 days ago
          Anonymous

          The fat you eat is the fat you wear. Enjoy wearing gasoline processed fat lmao

          • 4 days ago
            Anonymous
        • 4 days ago
          Anonymous

          >the decades of science that shows vegetables oils are a much healthier alternative to animal fats

        • 4 days ago
          Anonymous

          shut up and go back to plebbit with your lies and pseudoscience

    • 4 days ago
      Anonymous

      have a nice day now

  25. 4 days ago
    Anonymous

    Make olive oil your main added fat. Atherosclerosis is almost nonexistent on Med countries

    • 4 days ago
      Anonymous

      Make red meat your main protein and fat source. People from countries that hunt primarily for meat do not have high rates of heart attacks.

      So which one is it IST

      • 4 days ago
        Anonymous

        Both are right. In both cases it's the lack of PUFA.

      • 4 days ago
        Anonymous

        I am the olive oil guy. Let me say that meat is based too. Thank you.

    • 4 days ago
      Anonymous

      Those studies were paid for by olive oil manufacturers

      • 4 days ago
        Anonymous

        Yeah but olive oil is tasty. I even do pan fried chicken in it.

      • 4 days ago
        Anonymous

        >Assuming that """BIG OLIVE OIL""" exists like BIG PHARMA does
        >Assuming that there are a bunch of corporations that control Med countries exports
        >Assuming that farmers around the Mediterranean sea are employees of """BIG OLIVE OIL"""
        You have never step on a farm kid

        • 4 days ago
          Anonymous

          Its like you dont live in reality

        • 4 days ago
          Anonymous

          https://www.reuters.com/article/us-italy-crime-food-idUSKBN1602BD

          retard

          • 4 days ago
            Anonymous

            >reuters

            retard goy enjoy your early death from oxidized linoleic acid fucking up your cell linings causing early aging of your organs

  26. 4 days ago
    Anonymous

    Alcohol HIIT

    Go on a weeklong alcohol bender with minimal food and water to increase your blood alcohol levels to max, it will dissolve the fat deposits.

    Quit and take anti cholesterol meds to get rid of all the free floating fat before it clogs up again. Take a shitton of water

    • 4 days ago
      Anonymous

      The leanest and most athletic i've been was during one particular summer when all I did was drink lots of alcohol, eat little and lift/run/do lots of sports every day. I used to work a physical job too in a warehouse for constructions materials. Easiest the 1 period in my life when I felt the best, also the first gf I got was back then.

    • 3 days ago
      Anonymous

      >Go on a weeklong alcohol bender with minimal food and water to increase your blood alcohol levels to max, it will dissolve the fat deposits.
      "When you drink alcohol, it's broken down and rebuilt into triglycerides and cholesterol in the liver. So, drinking alcohol raises the triglycerides and cholesterol in your blood. If your triglyceride levels become too high, they can build up in the liver, causing fatty liver disease."

  27. 4 days ago
    Anonymous

    Make red meat your main protein and fat source. People from countries that hunt primarily for meat do not have high rates of heart attacks.

  28. 4 days ago
    Anonymous

    I clog them up purposefully to maximize my suffering so that I can transcend it by playing genshin impact and rolling for best girl yomi

  29. 4 days ago
    Anonymous

    A spoonful of rapeseed oil with every meal. It's a cheap and healthy alternative to olive oil with lots of Omega 3.

    • 4 days ago
      Anonymous

      lol olive oil isn't healthy because of it's omega 3's. It's healthy because its lack of Linoleic Acid. Rapeseed contains 3X more PUFA than olive, and less monounsaturated fats. You'd have to be a goddamn fool to purposely eat spoonfuls of garbage oils like that.

  30. 4 days ago
    Anonymous

    Keep inflammation to a minimum

  31. 4 days ago
    Anonymous

    test

  32. 4 days ago
    Anonymous

    Take Adestab and Vesugen and black garlic extract and Cayenne pepper.

  33. 4 days ago
    Anonymous

    >lifespan maxxs
    >dies in a car accident at 32

  34. 4 days ago
    Anonymous

    Unironically veganism, walking and plenty of sex

    I used to do a shitload of coke and booze tho...verdict's still out on how it has affected me

    • 4 days ago
      Anonymous

      You can always improve from where you are bro. Do your best

  35. 4 days ago
    Anonymous

    A whole food plant based diet not only prevents but actually reverses atherosclerosis.

    • 4 days ago
      Anonymous

      This is true but people don't want to accept this

  36. 4 days ago
    Anonymous

    Chelation therapy
    Not eating like a retard
    do you guys genuinely have problems with clogged arteries? I find it hard to believe that anyone who exercises regularly would have any issue with clogged arteries

  37. 4 days ago
    Anonymous

    Who cares, I'd rather die from a heart attack than cancer or dementia like people who look after their hearts.
    Enjoy dying slowly and painfully from cancer over a few year period, or slowly deteriorating from alzheimers until you are a vegetable in a nursing home.
    Meanwhile heart attack chads like me die instantly in their sleep.

    • 4 days ago
      Anonymous

      >cancer or dementia like people who look after their hearts.
      This isn't a thing so

  38. 4 days ago
    Anonymous

    Read this:

    https://academic.oup.com/cardiovascres/article/118/5/1188/6314360?login=false

    • 4 days ago
      Anonymous

      >https://academic.oup.com/cardiovascres/article/118/5/1188/6314360?login=false

      >Although small in magnitude, the reported reductions in relative risks

      >Funding
      The present analyses have been supported by a research grant from the ‘Barilla Center for Food and Nutrition Foundation (BCFN)’ within the framework of a project aimed at an evidence-based reformulation of the Food Pyramid for the prevention of CVD.

      Into the garbage it goes.

      Just a reminder there is no way of knowing which diet or foods are healthy unless:

      - hundreds of sets of identical twins from all different races are locked in a lab from birth to death. Every aspect of their entire lives are controlled and the same, such as sleep length and time, amount of sunlight, amount of stress, amount of sexual activity, amount and type of exercise, with just their diets being different. After weening they have to each eat two different diets for their entire lifespan, with the rest of their lives exactly the same. This study will last 100 years.

      Sorry not possible, so we will never know which diet is better and vegans will be arguing with carnivores forever like the losers they are.

      • 4 days ago
        Anonymous

        there were a bunch of RCTs in the 60s and 70s on institutionalized patients that avoided most of these issues. problem is they exonorated saturated fat and so were buried and or ignored

  39. 4 days ago
    Anonymous

    im vegetarian

  40. 4 days ago
    Anonymous

    It's all about inflammation.

    Keep you teeth, gum and gut healthy and if you should happen to fuck one of your teeth, don't get a root canal. ~~*Dentists*~~ will tell you that theyre safe but they'll cause chronic inflammation over years and years

  41. 4 days ago
    Anonymous

    I have heard that fiber and citrus bergamot can help lower bad cholesterol.
    Think there is any truth to this?

    • 4 days ago
      Anonymous

      >bad cholesterol
      No such thing

      • 3 days ago
        Anonymous

        >one study
        LDL-C doesn't cause atherosclerosis, it just leads to it inevitably unless you lower it to healthy levels with diet or drugs

        why are cholesterol denialists this fucking stupid

        • 3 days ago
          Anonymous

          It's not just one study, it's a review of many previous studies on cholesterol which the experts have found to have faulty conclusions.

  42. 3 days ago
    Anonymous

    Is butter bad? Eggs bad? My ldl skyrocket

  43. 3 days ago
    Anonymous

    >how to not get clogged arteries
    >reduce inflammation tm
    >how
    >just do it 4head
    thank you fit

    • 3 days ago
      Anonymous

      (not him)
      Unless you can edit your genes and immune system there's not a lot you can do besides get your genes mapped and eat a diet and live a lifestyle as consistent with that as possible. Which may not even be possible because of conflicting genes and food even their basic cultivars have changed so much since these genes proliferated the diet they benefited in may not even exist anymore. Most things beyond that are wishful thinking or clouded with bad information.

    • 3 days ago
      Anonymous

      Get more antioxidants in your diet and minimize the oxidant intake. That's the whole deal with sneed oils. Theyre very high in oxidizing fats. I can't remember from the top of my head if 3 or 6 was the oxidizing one. But even without direct intake of sneed oils modern diets have too much of the oxidizing fats and too few anti oxidants. Eat more tomato and tangerines. Also certain fatty fish.

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