so before is on keto and after is off keto, keto blasted my LDL well out of the normal values, our response ketosisters?
 UFOs Are A Psyop Shirt $21.68 |
 |
UFOs Are A Psyop Shirt $21.68 |
so before is on keto and after is off keto, keto blasted my LDL well out of the normal values, our response ketosisters?
Kys moxyte
it doesnt matter. native ldl is healthy. oxidised glycated ldl is not. your liver wouldnt go through the trouble of making it just to kill you
>Did keto
>Lost weight
Worked for me
>Worked for me
sshhh don't say that or else that redditor will be assmad
same
lost 132.277357 pounds most of which was after finding keto and stopped having weight loss plateaus by doing cico liek a fricking moron
didn't work out because I snapped my back, keto helped me lose 150 lbs but it's time to say goodbye because aint nobody want to do that shit forever
levels were good after dropping weight, surprised I didnt turn bald like you homosexuals said I would but working out is impossible on low carb and these gainez wont come by themselves
Remember when they lied for years about dietary cholesterol?
>dietary cholesterol doesn't affect cholesterol LOL MORON
>okay it does, but only HDL! FRICKING moron
>alright maybe LDL too, but not that much. homosexual
>alright okay a bunch, but LDL IS GOOD FOR YOU. IT'S TRIGLYCERIDES THAT KILL YOU AND THOSE AREN'T AFFECTED. DIPSHIT FRICKING FRICK FRICKING FRICKER
>okay triglycerides do frick you up... but uh... the reason americans are fat is because they eat too much spinach.... you're a israelite
I watched this happen in real time so don't even try.
Remember when Shawn Baker lied about fruit causing back pain?
Government lies about health?
Why would they do that?
Dietary cholesterol isn't associated with increased cardiac mortality in any population, and it also isn't associated with an increase in LDL cholesterol EXCEPT in 20-30% of the population classified as dietary cholesterol hyperresponders, in which both LDL and HDL increase proportionately in response to high dietary cholesterol.
>The evidence suggests that a diet including more eggs than is recommended (at least in some countries) may be used safely as part of a healthy diet in both the general population and for those at high risk of cardiovascular disease, those with established coronary heart disease, and those with T2DM
https://www.mdpi.com/2072-6643/7/9/5344/htm
>Epidemiological data do not support a link between dietary cholesterol and CVD
https://www.cambridge.org/core/services/aop-cambridge-core/content/view/S0007114511000237
>Consuming an egg-free NP diet for 12 weeks decreased LDL concentration (−0.3 ± 0.3 mmol/L) in older adults with overweight and obesity, while the HP diet, where at least 3 whole eggs were consumed per day, did not influence any marker of cardiometabolic health. Though contrary to our hypothesis, these results are consistent with recent findings [61,62,63] and show that consuming whole eggs does not influence total cholesterol, LDL concentrations [62], endothelial function, blood pressure [61], nor atherosclerosis [63] in at risk adults with overweight and obesity.
https://www.mdpi.com/2072-6643/10/7/946/htm
>Our patients [who ate 30-35 eggs per day over the course of the study] imbibed over 7,000mg of cholesterol per day. Contrary to expectations, serum cholesterol and lipoprotein levels remained normal throughout the period of the study.
https://www.jprasurg.com/article/0007-1226(75)90127-7/pdf
However, saturated fat increases LDL cholesterol by causing downregulation of LDLR (LDL receptor) which means there are less LDL receptors in hepatocytes to remove LDL-C from the blood
If cholesterol is bad why is your brain made out of it
Cholesterol is bad because your brain is made of it.
They want everyone to be nutritionally lobotomized good leftists.
Op here, the same bloodwork also showed my test levels going from 21nmol/L on keto to 13nmolL off...
Its over
Nice try, moxyte.
Ketogenic diet increases testosterone. Pic related.
that's exactly what he said you fricking mongoloid.
that post just proves that all you ketolards are fricking moronic
Thats why I posted bloodwork showing my test level going much lower when stopping keto, are you moronic?
I misread your post. My apologies.
If you are sincerely worried about cholesterol it's not a problem. More cholesterol is better. Always do the opposite of the mainstream advice.
moronic, because the people in those poor countries aren't dying of heart disease
they are dying of other causes
What was you LDL before keto?
Oh you dont know?
Vegan shill thread
Nah it's not a vegan shill. You can tell because he posted this:
A vegan would never let anyone know that cholesterol (only found in animal foods) and ketogenic diets raise testosterone.
Cholesterol is NOT the limiting factor of steroidogenesis. In men without damage to the testicles, the reason for lower testosterone is SECONDARY (having to do with the hypothalamus or pituitary not sending enough of a signal to the testicles). Not insufficient cholesterol or fat. That is to say that testosterone levels are regulated by brain signaling. The endogenous cholesterol production is more than enough to supply substrate for testosterone synthesis, hence why vegans have similar levels of testosterone to meat eaters despite eating little to no cholesterol.
Vegans have equal or higher testosterone levels:
https://pubmed.ncbi.nlm.nih.gov/32332862/
https://pubmed.ncbi.nlm.nih.gov/33599941/
For testosterone synthesis cholesterol requirements, healthy adult males produce ~14-42 micromoles testosterone per day:
https://www.ncbi.nlm.nih.gov/pubmed/14083165
One molecule of testosterone requires one molecule of cholesterol. Under gonadotropin stimulation, 700 million Leydig cells (however, adults can have up to 1573 million so this is a conservative estimate - pubmed.gov/3779804) synthesize ~13.2 micromoles per day when external sources are not provided:
https://www.ncbi.nlm.nih.gov/pubmed/736271
https://www.ncbi.nlm.nih.gov/pubmed/2226298
Meaning some plasma import is theoretically required. In humans, LDLR is the primary pathway for this, while SRB1 functions as a backup:
https://www.ncbi.nlm.nih.gov/pubmed/20515451
LDLR kinetics saturate at ~25mg/dL plasma LDL-C:
https://www.ncbi.nlm.nih.gov/pubmed/198627
whereas SRB1 kinetics saturate at ~30 mg/dL plasma HDL-C:
https://www.ncbi.nlm.nih.gov/pubmed/10872459
What that means is testosterone production is not substrate-limited unless there is extremely low blood cholesterol levels, achievable only with drugs and/or rare genetic disorders, not dietary cholesterol restriction. The human liver alone (the intestine being the other source of LDL and HDL) is capable of endogenously synthesizing ~2.6 micromoles of cholesterol per kg bodyweight per day, more than enough when considering ~70% circulating LDL-C remains unused and is simply taken up by the liver again for recycling:
https://www.ncbi.nlm.nih.gov/pubmed/8245716
Testosterone production in adult males is 4-12 mg/day, while endogenous cholesterol synthesis in humans is 12-13 mg/kg a day. The receptors responsible for delivering cholesterol to Leydig cells are saturated at 50 mg/dL and 30 mg/dL blood LDL-C and HDL-C, respectively. You have more than enough:
http://science.sciencemag.org/content/232/4746/34
http://science.sciencemag.org/content/271/5248/518
Over and above this is actually bad, for the cells can become overloaded with cholesterol or it can start to oxidize, both of which are toxic and impair production:
http://www.sciencedirect.com/science/article/pii/S002191509900310X
http://www.jbc.org/content/288/16/11509.short
Not to mention carbs are critical to have high testosterone because of SHBG, this is a protein that binds to testosterone and renders it inert. Eating carbohydrate blocks SHBG synthesis in the liver because of the increases in insulin which increases free testosterone. The more insulin you have over the course of the day, the lower the SHBG. Also, ketosis increases cortisol which reduces testosterone biosynthesis.
High SHBG is a common problem among carnivore diet practitioners. Their terrible blood work is a lot of the reason many such as Paul Saladino have started to embrace eating carbs.
The guy in your pic (Vince Gironda) died of a heart attack. Having big muscles doesn't mean your arteries aren't clogged.
>TL;DR: testosterone levels are controlled by the brain, cholesterol is not the issue, higher cholesterol levels will not give you higher testosterone.
LDL is good
It fuels your brain.
High LDL doesn't matter if you're not fat.
Just don't be fat.
it does matter, why would it not matter if you're not fat?
Because it's not correlated with medical issues if you're not fat.
it doesnt. only drug pushers say otherwise. youve been fooled
who's pushing drugs? the patents for statins ran out, they're generic drugs, pharma doesn't make shit
the new hot drugs are PCSK9 inhibitors which lower LDL without the side effects of statins
>without the side effects of statins
dying with a lowered cholesterol is not a successful intervention
>deaths of cardiac origin were numerically higher in the evolocumab group than in the placebo group in the FOURIER trial
https://pubmed.ncbi.nlm.nih.gov/36585131/
>so before is on keto and after is off keto, keto blasted my LDL well out of the normal values, our response ketosisters?
Ket is hypersanguinic, hot and wet. Could work if you're starting out phlegmatic with lots of inner cold, and not much inner moisture, won't work if you're one of the diabetic fat guys.
t. humoral pathology pro