Is it testosterone or estrogen that ages you more ?
Or is it an extreme imbalance of one ?
Is it just better to have a proper healthy balance based on your sex ?
Is it testosterone or estrogen that ages you more ? Or is it an extreme imbalance of one ?
test ages you, est de-ages you and also gives you youthful features. well known result among trannies.
Checked…….
So if your just a healthy man and get lots of Vit D etc. that results in healthy test levels this will age you ?
So being a healthy man that results in healthy test levels ages you ?
Breh
I’m wondering if one of he reasons Asians age so well is all of the onions they eat idk.
Onions they eat*
ONIONS THEY EAT****
50Y
S.0.Y
I think our bodies are just sensitive to androgen so much that it activates autoimmune disorders like bad skin and hair falling out etc. I guess high testosterone is bad after all.
Is it possible that androgens aren’t supposed to be harmful and it’s a weak/compromised immune system that’s allowing them to be harmful ?
yeah bro you just need to take vitamin c sups with your test injections and youll be fine
Then we all collectively have shitty immune systems. But look at bodybuilders after hopping on trt and roids they age 10 years in 1.
DHT ages you the most while testosterone is neutral and estrogen is beneficial.
Estrogen isn't benefical
>protects against cholesterol
>increases bone mass
>makes the skin look younger
>promotes hair growth
DHT is the one that ages you, e2 is actually good for your skin, heart, etc. in moderation
So Kevin from haircafe is right about everything and it’s a trash hormone ?
What about impotence due to low DHT or is that just because DHT blockers also lower test ?
>So Kevin from haircafe is right about everything and it’s a trash hormone ?
The only use for DHT is in the brain and fin doesn't inhibit 5ar1 while dutasteride can't cross the BBB.
>What about impotence due to low DHT or is that just because DHT blockers also lower test ?
5ar inhibitors will change your testosterone and estrogen levels. If the ratio changes too much, it can lead to side effects.
>fin doesn't inhibit 5ar1 while dutasteride can't cross the BBB.
What does this mean in simple terms ? I don’t know the euphemisms
Neither drug affects your brain at all, nor anything else for that matter. All you need to watch out for is your testosterone to estrogen ratio.
>So Kevin from haircafe is right about everything and it’s a trash hormone ?
He's going a bit overboard. Some DHT is good, flatlining your DHT will cause side effects in some people. All you really want/need is to decrease your DHT to below a certain threshold.
>What about impotence due to low DHT or is that just because DHT blockers also lower test ?
I assume you're talking about fin/dut. They aren't DHT blockers, they're 5a-reductase inhibitors (5a-reductase is the main chemical that turns testosterone into DHT).
If you take finasteride [1mg/day] the scientific data was that you decrease DHT by 70%, increase testosterone by 10-20%, and also increase E2 [estradiol] by 10-20%.
This is a pretty good trade for 95%+ of people but really sucks for the people who are already high E, and that extra 10-20% puts them out of range, leading to all kinds of shit (problems with libido, problems with erections, gynocomastia risk, etc.).
If your E2 is high, the best way to lower it is to cut, fat is where most aromatase goes on. Less fat => less aromatase => less E2.
What is E,E2 ?
So you’re saying the skinnier you are the lest test you have ? The less fat you are etc.
any thoughts on stuff like saw palmetto ?
E = estrogen (all types)
E1 = Estrone
E2 = Estradiol (THIS IS THE IMPORTANT ONE)
E3 = Estriol
When people are talking about 'estrogen', they really mean estradiol (E2).
Fat guys have higher estrogen than skinny guys because they have more fat, and fat is where most of the conversion from testosterone to estradiol goes on.
>saw palmetto
isn't backed up by science [yet?], allegedly it's a 5ar inhibitor so it would work literally the exact same way as fin/dut... which is really funny because people on IST shill it and in the same post say that finasteride is a troony pill lmao
So you would want low E2 when taking fin to stop side effects is what you’re saying ?
Or just high test in general to offset the reduction
If your E2 is normal (95%+ chance that it is), you won't have sides from finasteride.
If your E2 is high, the easiest way to fix it is to stop being fat.
>Some DHT is good
Fin leaves you with 30% of DHT while dut leaves you with around 5%. So even if there was some theoretical need we haven't discovered yet, there's still enough DHT around in your system.
Otherwise good post.
dut and fin both destroy your sperm like crazy. you fingays massively overplay how little damage that shit does to your entire hormone system.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3205531/
>In a study, a 48-year-old man on finasteride found a decrease in his sperm DNA fragmentation index (DFI) by 9% three months after discontinuing the drug and a further fall of about 5% in the subsequent three months.[1] Similar improvement was observed in two other cases of azoospermia and severe oligospermia, 6 months after discontinuing finasteride.[2]
>A patient on finasteride (1 mg) for 1 year, diagnosed with oligospermia (impaired spermatogenesis) 5 years earlier, was found to have increased semen volume immediately after cessation of finasteride and his sperm concentration improved to more than 10 × 106/ml about 4 months later.
So perfectly reversible over time.
And the 1mg dose didn't even seem to have any effect at all.
>A study reported that spermatogenesis is not adversely affected by finasteride (at 1 mg dose), although the same study observed that the ejaculate volume, prostate volume, and serum PSA showed a decrease with finasteride administration.[7]
>Finasteride does not have negative effects on erectile function, as established by many studies using international index of erectile function-5 questionnaire and nocturnal penile tumescence testing.
Good to know.
Your study can be tl;dr'd as finasteride/dutasteride may have a slight negative impact in fertility (especially at higher doses) but that effect is perfectly reversible upon cessation.
The impact on sperm quality is low, reversible and generally subsides over time.
>In both treatment groups, total sperm count, compared with baseline, was significantly decreased at 26 wk (D, -28.6%; F, -34.3%) but not at 52 wk (D, -24.9%; F, -16.2%) or the 24-wk follow-up (D, -23.3%; F, -6.2%). At 52 wk, semen volume was decreased (D, -29.7%; F, -14.5%, significantly for D) as was sperm concentration (D, -3.2%; [corrected] F, -7.4%, neither significant). There was a significant reduction of -6 to 12% in sperm motility during treatment with both D and F and at follow-up. Neither treatment had any effect on sperm morphology.
https://pubmed.ncbi.nlm.nih.gov/17299062/
incels have low test and they all look 20 in their 30's with pristine skin and perma youthfulness etc.
unironically
> Is it just better to have a proper healthy balance based on your sex ?
This. It is the imbalances that frick things up. Stay away from xenoestrogens.
Broscience
Uhhh pretty sure neither age you. It’s your cells wearing down and becoming less useful over time